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The overexpression of arginine methyltransferase 5 (PRMT5) in solid and hematological tumors leads to the elevation of the methylation level of arginine residues on functionally related proteins in tumor cells, which affects cell cycle regulation, mRNA splicing, cell differentiation, signal transduction, and other physiological processes

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Pool the peak fractions containing the protease and measure the volume

2000 Retinoblastoma protein dephosphorylation is an early event of cellular response to prooxidant conditions FEBS Lett